A 1989 article in the New England Journal of Medicine (April 6, pp. 904-909) presented the following all-cause death rates for the corresponding cholesterol levels:

Lipids, in conjunction with the immune system, play a critical role in the body's defense mechanism. Cholesterol, triglycerides and estrogen are "anabolic" lipids in the lipidic defense. Fatty acids, such as leukotrienes and testosterone are "catabolic" lipids in the lipidic defense. To learn more about the "diphasic lipidic defense" (anabolic and catabolic lipids) see the clinical research of Emanuel Revici MD.*

Catabolic lipids are the first line of defense. And once the catabolic lipids have done their job (mopped up the torn tissue or killed the pathogen or cancer cell, etc.) they must be neutralized. If they are not neutralized they will go on "mopping up" and "killing" normal cells, tissues, glands and organs in the body.

Anabolic lipids neutralize catabolic lipids. Low cholesterol, an anabolic lipid, means excess catabolic lipids. Excess catabolic lipids result in excess free radical activity. Excess free radical activity means damage to cells, tissues, glands and organs. Therefore, Tissue & Cell Cholesterol determines how much DAMAGE you can take before it kills you!!!

by Lynne August

Comment: Specific lipids, in conjunction with the immune system, play a critical role in the body's defense mechanism. Cholesterol is one of these lipids. Hence, the lower the cholesterol the greater the compromise in the defense and the higher the death rate from all causes. Ever since high cholesterol has been associated with heart disease there is the erroneous assumption that "the lower the cholesterol the better." This study provides statistical evidence to the contrary.

© Health Equations 2002

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